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Angibloc (Losartan)

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Angibloc is a medication mainly used to treat high blood pressure. It is also used for diabetic kidney disease, heart failure, and left ventricular enlargement. It is taken by mouth. It may be used alone or in addition to other blood pressure medication.

Other names for this medication:
Angibloc, Cormac, Cozaarex, Faxiven, Fensartan, Fortzaar, Gitox, Klosartan, Lavestra, Lifezar, Loctenk, Lorista, Lortaan, Losadel, Losamet, Losardil, Losart, Losartan, Losartas, Losartil, Losazide, Lostad, Lotar, Loxibin, Lyosan, Myotan, Osartil, Ostan, Portiron, Psycholanz, Sarvas, Tensaar, Tensiohess

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Cozaar, Calan, Cordarone, Lopressor, Aceon, Lanoxin, Microzide

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Also known as: Losartan


Angibloc (losartan potassium) is an angiotensin II receptor blocker acting on the AT1 receptor subtype. Losartan potassium, a non-peptide molecule, is chemically described as 2-butyl-4-chloro-1-[p-(o-1Htetrazol- 5-ylphenyl)benzyl]imidazole-5-methanol monopotassium salt.

Angibloc is a white to off-white free-flowing crystalline powder with a molecular weight of 461.01. It is freely soluble in water, soluble in alcohols, and slightly soluble in common organic solvents, such as acetonitrile and methyl ethyl ketone. Oxidation of the 5-hydroxymethyl group on the imidazole ring results in the active metabolite of losartan.

Angibloc is available as tablets for oral administration containing either 25 mg, 50 mg or 100 mg of losartan potassium and the following inactive ingredients: microcrystalline cellulose, lactose hydrous, pregelatinized starch, magnesium stearate, hydroxypropyl cellulose, hypromellose, and titanium dioxide.

Angibloc 25 mg, 50 mg and 100 mg tablets contain potassium in the following amounts: 2.12 mg (0.054 mEq), 4.24 mg (0.108 mEq) and 8.48 mg (0.216 mEq), respectively. Angibloc 25 mg, Angibloc 50 mg, and Angibloc 100 mg may also contain carnauba wax.


Take Angibloc tablets orally with or without food. Do not crush or chew it. Take Angibloc once or twice a day at the same time. If you want to achieve most effective results do not stop taking Angibloc suddenly.


If you overdose Angibloc and you don't feel good you should visit your doctor or health care provider immediately. Symptoms of Angibloc overdosage: fainting, feeling lightheaded, rapid heartbeat.


Store at room temperature between 15 and 30 degrees C (59 and 86 degrees F) away from moisture, light and heat. Keep container tightly closed. Throw away any unused medicine after the expiration date. Keep out of the reach of children in a container that small children cannot open.

Side effects

The most common side effects associated with Angibloc are:

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Side effect occurrence does not only depend on medication you are taking, but also on your overall health and other factors.


Do not take Angibloc if you are allergic to Angibloc components. Do not take Angibloc if you're pregnant or you plan to have a baby, or you are a nursing mother. Angibloc can harm your baby. Do not use Angibloc if you are taking salt substitutes or potassium supplements, other blood pressure medicine, diuretic (water pill). It can be dangerous to use Angibloc if you suffer from or have a history of liver disease, kidney disease, heart failure. If you want to achieve most effective results without any side effects it is better to avoid alcohol. Avoid machine driving. Do not stop taking Angibloc suddenly.

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Compressive and diametral tensile strength of composite restorative resins. Dye-free determination of the focalization position for the hollow fiber flow field flow fractionation (HF5) of proteins. Patients were identified from the trauma registry using DRG International Classification of Diseases-9th Rev. QUICKI is considered more sensitive than HOMA-IR in differentiating simple steatosis from normal liver histology. We asked: (1) Does percutaneous calcium phosphate injection improve validated patient-reported outcome measures?. We investigated the feasibility and validity of the 5-minute National Institute of Neurological Disorders and Stroke and Canadian Stroke Network (NINDS-CSN) protocol and six-item screener (SIS) in stroke patients by telephone administration. This paper reviews natural products as antidepressants documented in Chinese patents so far.

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Anticholinergic burden quantified by anticholinergic risk scales and adverse outcomes in older people: a systematic review. A zebrafish cDNA was obtained that presumably codes for a 438 amino acid ovarian TNF receptor (OTR) that was identified as a death-domain-containing member of the TNF receptor family. The effect of the extract of Crocus sativus and its constituent, safranal on inflammatory markers in sensitized guinea pigs was examined. The floral repressor gene FLOWERING LOCUS C (FLC) in Arabidopsis thaliana is a useful system to address these questions. CE is effective at excluding clinically significant anastomotic problems, especially after clinical anastomotic leaks. Myelodysplasia produces a neurological lesion with unpredictable urodynamic sequelae. Structural Requirements of Histone Deacetylase Inhibitors: SAHA Analogs Modified on the Hydroxamic Acid. In a multivariate analysis the higher use of prescribed analgesics and antidepressants was explained almost entirely by a higher morbidity in the minority study groups. Paired human fibrosarcoma cell lines that possess or lack endogenous mutant N-ras alleles as experimental model for Ras signaling pathways. Individual clinicians should avoid making clinical decisions on the basis of relative reductions of morbidity or mortality, should use evidence-based clinical practice guidelines rather than those based on authority whenever possible, should make use of patient-information material and, most important, should have a consistent policy of obtaining informed consent from patients before they participate in potentially harmful preventive programs.

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This review focuses on proteomic techniques and their applications which have advanced our understanding of the signaling mechanisms involved in CVDs at the levels of protein complex/protein-protein interaction, post-translational modifications and signaling induced protein changes. CALHM1 deficiency impairs cerebral neuron activity and memory flexibility in mice. The current model of hepatitis C virus (HCV) production involves the assembly of virions on or near the surface of lipid droplets, envelopment at the ER in association with components of VLDL synthesis, and egress via the secretory pathway. Sixty patients between the ages of 1 to 18 years were analyzed for the presence of KIAA1549-BRAF fusion gene products using reverse transcription-PCR and benicar to losartan dose sequencing. GaAs coupled micro resonators with enhanced sensitive mass detection. Our results will facilitate evidence-based management of patients with LBP and identify key areas for future research.

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